Contact Us
+888 976 6272
한국어
    Sign InRegisterSign Out
    My Account
    Product was added to your cart

    Cart

    Bone is living, growing tissue. Throughout life, our bodies are breaking down old bone cells and rebuilding new bones in a continuous cycle (bone remodeling). We gain bone by building more bone cells than we lose.After about age 40, however, this balance is typically reversed, with bone loss occurring at a much faster rate than is replaceable, leaving our bones brittle and leading to osteoporosis with increased risks of fracture, particularly of the hip, spine, wrist and shoulder.

    Osteoblasts and osteoclasts are types of cells the human body uses to repair broken bones. Osteoclasts break down old bone tissue allowing osteoblasts to replace it with new material. Together, these cells facilitate bone mending and bone growth. However, as we age osteoblast slows down and bone mineral density continues to decrease, making our bones more susceptible to fracture.

    Losing more than 25% of bone mass is enough for a clinical diagnosis of osteoporosis. In the US, 1/3 of women and 1/4 of men have the disease, with the figure increasing each year. What may be the cause of all this?

    OSTEOPOROSIS WEAKENS BONES
    THE 4 FUNCTIONS OF SAC CALCIUM
    SAC AS HORMONE REGULATOR

    Bone metabolism is controlled by the interaction of a number of hormones.

    Hormonal imbalance can wreak havoc on bone metabolism. SAC helps triggering hormones to maintain bone metabolic balance, by signaling pituitary hormones to trigger the release of thyroid and parathyroid hormones.

    SAC AS CALCIUM NAVIGATOR

    Inactive calcium molecules such as protein-bound calcium do not know where to go.

    SAC navigates calcium to reach its ultimate destination – the bone. SAC calcium will not be deposited in the wrong places such as the kidneys or blood vessels.

    SAC AS CALCIUM ACTIVATOR

    About 50% of calcium in our blood is inactive.

    SAC’s ionization process activates inactive calcium molecules to be used in bone building by stimulating the secretion of thyroid hormone (TH), which is responsible for depositing minerals in our bones.

    SAC AS BONE MINERAL BUILDER

    Without any side effects, SAC calcium considerably aids in the prevention and treatment of osteoporosis, thereby reducing fracture risks by significantly improving bone mineral density.

    Within a relatively shorter period of time, SAC helps to deposit other essential minerals from one’s diet into the bone along with calcium for healthier bones.

    SERIOUS SIDE EFFECTS OF PRESCRIPTION DRUGS
    Dangerous & More Disappointing!

    Physicians typically recommend prescription medications for the treatment of osteoporosis, but these drugs are notorious for their dangerous side effects.

    These drugs are designed to “increase” bone density by retaining dead bone mass through the inhibition of osteoclasts, the cells that are responsible for natural bone resorption, which provides calcium for our body’s needs.

    Our body takes calcium from bones – even at the risk of bone fractures, because of calcium’s urgent and crucial role in healthy cell replication, neurotransmission, heart functions, and endocrine functions.

    Disrupting essential bone metabolism may lead to serious health hazards, which is why there are so many reports that osteoporosis drugs actually make bones more brittle and that people who are treated with these drugs tend to suffer more bone fractures.*

    If our bones don’t provide the required calcium right away, many of our body functions suffer, resulting in serious conditions.

    Treated with those drugs, limited space within the bones is taken up by dead or nearly-dead bone mass, preventing new bone mass from being created. As a result, bones become more fragile and vulnerable to fracture!

    It won’t be a surprise to see a modest increase in density from bone scans performed on those treated with these drugs, which promotes a false sense of security based on all the useless dead bone mass being counted, preventing patients from seeking alternative treatments until it’s too late.

    “But research has increasingly linked bisphosphonates to less common but more serious and long-lasting problems, including an abnormal heart rhythm (atrial fibrillation); incapacitating bone, joint, and muscle pain; and bone loss in the jaw (osteonecrosis). Some evidence suggests long-term use may actually increase the risk of a certain type of fracture.”
    TWO CATEGORIES OF PRESCRIPTION DRUGS
    Anti-Resorptive Therapy & Anabolic Therapy

    1. ANTI-RESORPTIVE DRUGS

    Drugs that slows bone loss

    Antiresorptive therapies are used to increase bone strength by slowing or stopping osteoclast in individuals with osteoporosis and include five principal classes of agents: bisphosphonates, estrogens, selective estrogen receptor modulators (SERMs), calcitonin and monoclonal antibodies such as denosumab.

    Bisphosphonates are the most commonly prescribed antiresorptive medications and remain first-line treatment for osteoporosis. Bisphosphonates are adsorbed into the mineralized surface of bone and are internalized by osteoclasts, interfering with biochemical processes involved in bone resorption; they also induce apoptosis of osteoclasts.

    Denosumab, the first biologic introduced for osteoporosis treatment, is a fully human monoclonal RANKL antibody, and by binding to RANKL, it prevents the binding of RANKL to RANK; this leads to inhibition of osteoclast activation and function.

    Artificially stopping osteoclast which is a natural process our body uses to provide needed calcium for our body’s essential functions, becomes a precursor to manifold health problems. Inhibiting osteoclast to maintain the bone density may cause bigger problems than preventing fractures. SAC utilizes both osteoblast and osteoclast to build healthy bones and naturally prevents fractures.

    2. ANABOLIC DRUGS

    Drugs that increase the rate of bone formation

    Trailing the development of antiresorptives for osteoporosis is the development of anabolic agents designed to increase bone mineral density (BMD) by stimulating bone formation, osteoblast. Sodium fluoride was a promising anabolic agent for the treatment of postmenopausal osteoporosis, but it was found to increase the risk of nonvertebral fractures despite dramatic increases in BMD and is not approved by FDA in the USA. GH (growth hormone) has also been the object of interest as an anabolic agent for the skeleton. PTH (parathyroid hormone), long known to have anabolic potential was “rediscovered” about 15 yr ago, but the bone building effect was not very impressive (only 6% increase) and also came with many side effects. The statins, cornerstones of lipid-lowering therapy, have also recently been revisited as potentially important skeletal anabolic agents, but we are too familiar with the side effects of statins.

    Parathyroid hormone (PTH) and its analogue, teriparatide (Forteo) may cause unwanted side effects that require medical attention. Also, because of the way these medicines act on the body, there is a chance that they might cause other unwanted effects that may not occur until months or years after the medicine is used. These delayed effects may include certain types of cancer, such as bone cancer. More common effects include abdominal pain, confusion, constipation, depression, headache, incoherent speech, vomiting, etc.

    Calcilytics increases the secretion of parathyroid hormone (PTH), which has a temporary anabolic effect on bone tissue, producing an increase in both bone volume and bone density due to increased bone deposition and resorption. However, long-term use of these causes resorption, degrading the bone to raise blood calcium. Consequently, these drugs have been researched for the treatment of osteoporosis, though with only limited success.

    SAC therapy triggers TH (Thyroid Hormone), not PTH (Parathyroid Hormone), in building bones, the way our body does it naturally! Nothing works more naturally without side effects than SAC therapy.

    Current osteoporosis drugs in the market do not increase bone mineral density in any significant way even after prolonged use. Moreover, new studies found that dead bone mass maintained by these drugs make bones unhealthy and more fragile. Our Body takes calcium from our bones for the use of essential body functions and therefore, artificially blocking calcium supply from our bones will cause a cascade of bigger health problems. SAC calcium, on the other hand, triggers natural bone forming process by restoring healthy calcium homeostasis.

    THE CLINICAL RESULTS OF SAC
    HUMAN TRIAL RESULT

    Above indicates real bone mineral density (BMD) increase of 49 year-old male since taking SAC calcium. Measurement shows a significant increase of BMD (T-score of 1.5) over a period of 6 months. BMD is measured by FDA approved ultrasound bone densitometer by BeamMed.

    ANIMAL TRIAL RESULT
    Lab Animal Test 2011: 27(4), 301-307

    Effects of SAC Calcium on bone turnover and calcium balance in ovariectomized rats.
    Results reveal that the BMD of rats with induced osteoporosis has returned to normal with SAC intake. Please click the link below for the full research paper.

    RESEARCH LINK for Full Study : SAC Calcium Effects on Overiectomized Rats

    About Us

    Marah Natural is dedicated to improving the lives of patients with ongoing clinical trials and research. We are also focused on developing high quality supplements that delivers satisfying results.

    Marah Natural USA
    Queens, NY 11358, USA
    info@marahnaturalusa.com
    (888) 976 6272

    FDA – COMPLIANCE – DISCLAIMER

    The information on this website has not been evaluated by the Food & Drug Administration or any other medical body. We do not aim to diagnose, treat, cure or prevent any illness or disease. The Information shared on this website and affiliated pages is for educational purposes only. You must consult your doctor or healthcare professional before acting on any content on this website, especially if you are pregnant, nursing, taking medication or have a medical condition. Individual articles are based upon the opinions of the respective author, who retains copyright as marked. The information on this website is not intended to replace a one-on-one relationship with a qualified health care professional and is not intended as medical advice. Marah Natural encourages you to make your own health care decisions based upon your research and in partnership with a qualified health care professional.

    Copyright © 2019 Marah Natural USA. All rights reserved.